Aaron Grossberg, MD PhD


Assistant Professor


Radiation Medicine


Radiation Medicine, Neuroscience, Physiology, Neuroinflammation




BA in Psychology, University of Colorado at Boulder, summa cum laude


PhD in Neuroscience, Neuroscience Graduate Program, Oregon Health and Science University

Thesis: Inflammmatory Regulation of Hypothalamic Neurons in Sickness Behavior

MD: Oregon Health and Science University School of Medicine, magna cum laude


Radiation Oncology, The University of Texas MD Anderson Cancer Center


2016 RSNA Research and Education Foundation Research Fellow Award

2015 –4th Annual Dr. Mary Fletcher Memorial Award, MD Anderson Cancer Center

2013 –Rubenstein Radiation Research Scholarship, Oregon Health and Science University

2009 –Ruth L. Kirchstein National Research Service Award (F30)

2008-2010 - N.L. Tartar Research Fellow

2007 –OBI/Doernbecher Children's Hospital Foundation Pediatric Neurobiology of Disease Fellow


Association of Body Composition With Survival and Locoregional Control of Radiotherapy-Treated Head and Neck Squamous Cell Carcinoma.

Dose painting with Gamma Knife: Two techniques for delivering different doses to areas of recurrent or residual tumor after resection of brain metastases.

Cancer- and endotoxin-induced cachexia require intact glucocorticoid signaling in skeletal muscle.

Hypothalamic signaling in anorexia induced by indispensable amino acid deficiency.

Expression of myeloid differentiation factor 88 in neurons is not requisite for the induction of sickness behavior by interleukin-1β.

Central nervous system inflammation induces muscle atrophy via activation of the hypothalamic-pituitary-adrenal axis.

Inflammation-induced lethargy is mediated by suppression of orexin neuron activity.

Hypothalamic mechanisms in cachexia.

Arcuate nucleus proopiomelanocortin neurons mediate the acute anorectic actions of leukemia inhibitory factor via gp130.