OHSU

Kristina Young, MD, PhD

kyoungTitle:

Resident

Department:

Radiation Medicine

Specialty:

Radiation Medicine

Residency:

Oregon Health & Science University, Portland, Oregon

Resident Year:

PGY-5, 2014 - 2015, Oregon Health & Science University, Portland, OR
PGY-2 (1st year Radiation Medicine) in 2011

Internship:

Internal Medicine Preliminary Year (PGY1), Providence St. Vincent’s Medical Center, Portland, OR, 2010-2011

Undergraduate:

BS, Chemical Engineering with Bioengineering Emphasis, University of California Los Angeles, Los Angeles, CA, 2003

Medical School:

MD, PhD, Oregon Health & Science University, Portland, OR, 2010

Interest and Accomplishments:

Head and Neck cancer, Tumor biology, Targeted Molecular Therapy.
Rubinstein Radiation Research Scholar.
RSNA Research & Education Foundation’s Research Medical Student Grant.
Albert M. Kligman Fellowship Recipient.
Appointment to T32 Pediatrics Training Grant.
Tartar Trust Fellowship Recipient

Hobbies:

Soccer, Tennis, Kickboxing, Hiking, Snowshoeing, Traveling, Baking, Jogging, Swimming, Bicycling, Happy Hour.

School of Medicine Hooding Cermony 2010:

View photo

Publications:

  • Hoot K, Oka M, Han G, Zhang Q, Wang XJ. HGF/c-Met upregulation contributes to angiogenesis associated with epithelial Smad2-deletion.
  • Hoot, K.E., Li, A.G., Lu, S.L., Han, G., Ju, W., Bottinger, E.P., Wang, X.J. Keratinocyte-specific Smad2 ablation results in increased epithelial-mesenchymal transition during skin cancer formation and progression.
  • Journal of Clinical Investigation, 2008 Aug;118(8):2722-32
  • Bornstein S, Hoot K, Han GW, Lu SL, Wang XJ. Distinct roles of individual Smads in skin carcinogenesis. Mol Carcinog. 2007 Aug;46(8):660-4
  • Li AG, Lu SL, Han G, Hoot KE, Wang XJ. Role of TGFbeta in skin inflammation and carcinogenesis. Mol Carcinog. 2006 Jun;45(6):389-96

Conference Presentations:

  • Presented Data at Poster Session of The 100th Annual Meeting of the American Association for Cancer Research, Smad2 and HMGA2 coordinate to regulate Snail expression in skin squamous cell carcinomas (4/2009).
  • Oral Presentation at the Annual Society for Investigative Dermatology. Keratinocyte- specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion (5/2007).
  • Oral Presentation in the Western Society of Pediatric Research. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis (2/2007).
  • Presented Data at Poster Session of The International Skin Carcinogenesis Conference. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis (11/2006).
    (2/2006) Presented Data at Poster Session of The Timberline Symposium on Epithelial Cell Biology. Discerning the Spatial Effects of TGF?1 Overexpression on Inflammation and Carcinogenesis.
  • Hoot K, Zhang Q, Wang XJ. Smad2 and HMGA2 coordinate to regulate Snail expression in skin squamous cell carcinomas, 100th Annual Meeting of the American Association for Cancer Research, Proceedings of the AACR (2009) #1522.
  • Oka M, Hoot K, Wang XJ. Smad2 loss causes increased angiogenesis during skin carcinogenesis via HGF upregulation. 100th Annual Meeting of the American Association for Cancer Research, Proceedings of the AACR (2009) #4018.
  • KE Hoot, AG Li, S Lu, G Han, W Ju, EP Bottinger and X Wang. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion. Journal of Investigative Dermatology (2007) 127, S19.
  • Hoot, K.E., Li A.G., Lu S, Han G, Ju W, Bottinger E.P. and Wang X.J. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis. Journal of Investigative Medicine, Vol.55 (1), #395, (2007).