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Meet the Staff
Kristina Young, MD, PhD
http://myprofile.cos.com/khoot
Photo:
Hooding Ceremony
Title:
Resident
Department:
Radiation Medicine
Specialty:
Radiation Medicine
Resident Year:
PGY-2 (1st year Radiation Medicine) in 2011
Internship:
Internal Medicine Preliminary Year (PGY1), Providence St. Vincent’s Medical Center, Portland, OR, 2010-2011
Undergraduate:
BS, Chemical Engineering with Bioengineering Emphasis, University of California Los Angeles, Los Angeles, CA, 2003
Medical School:
MD, PhD, Oregon Health & Science University, Portland, OR, 2010
Interest and Accomplishments:
Head and Neck cancer, Tumor biology, Targeted Molecular Therapy.
3/2009 Rubinstein Radiation Research Scholar
3/2009 RSNA Research & Education Foundation’s Research Medical Student Grant
5/2007 Albert M. Kligman Fellowship Recipient
9/2006 Appointment to T32 Pediatrics Training Grant
5/2006 Tartar Trust Fellowship Recipient
Hobbies:
Soccer, Tennis, Kickboxing, Hiking, Snowshoeing, Traveling, Baking, Jogging, Swimming, Bicycling, Happy Hour.
Publications:
Hoot K, Oka M, Han G, Zhang Q, Wang XJ. HGF/c-Met upregulation contributes to angiogenesis associated with epithelial Smad2-deletion.
Hoot, K.E., Li, A.G., Lu, S.L., Han, G., Ju, W., Bottinger, E.P., Wang, X.J. Keratinocyte-specific Smad2 ablation results in increased epithelial-mesenchymal transition during skin cancer formation and progression. Journal of Clinical Investigation, 2008 Aug;118(8):2722-32
Bornstein S, Hoot K, Han GW, Lu SL, Wang XJ. Distinct roles of individual Smads in skin carcinogenesis. Mol Carcinog. 2007 Aug;46(8):660-4
Li AG, Lu SL, Han G, Hoot KE, Wang XJ. Role of TGFbeta in skin inflammation and carcinogenesis. Mol Carcinog. 2006 Jun;45(6):389-96
Conference Presentations:
4/2009 Presented Data at Poster Session of The 100th Annual Meeting of the American Association for Cancer Research, Smad2 and HMGA2 coordinate to
regulate Snail expression in skin squamous cell carcinomas.
5/2007 Oral Presentation at the Annual Society for Investigative
Dermatology. Keratinocyte-
specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion.
2/2007 Oral Presentation in the Western Society of Pediatric Research. Keratinocyte-specific
Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis.
11/2006 Presented Data at Poster Session of The International Skin Carcinogenesis Conference.
Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis.
2/2006 Presented Data at Poster Session of The Timberline Symposium on Epithelial Cell Biology.
Discerning the Spatial Effects of TGF?1 Overexpression on Inflammation and Carcinogenesis.
Hoot K, Zhang Q, Wang XJ. Smad2 and HMGA2 coordinate to regulate Snail expression in skin squamous cell carcinomas, 100th Annual Meeting of the American Association for Cancer Research, Proceedings of the AACR (2009) #1522.
Oka M, Hoot K, Wang XJ. Smad2 loss causes increased angiogenesis during skin carcinogenesis via HGF upregulation. 100th Annual Meeting of the American Association for Cancer Research, Proceedings of the AACR (2009) #4018.
KE Hoot, AG Li, S Lu, G Han, W Ju, EP Bottinger and X Wang. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion. Journal of Investigative Dermatology (2007) 127, S19.
Hoot, K.E., Li A.G., Lu S, Han G, Ju W, Bottinger E.P. and Wang X.J. Keratinocyte-specific Smad2 ablation promotes skin carcinogenesis via affecting genes related to tumor invasion and angiogenesis. Journal of Investigative Medicine, Vol.55 (1), #395, 2007.
Links of Interest:
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